Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.

Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.