Deprecated!
This application is deprecated. Please use the latest version of the tool here.
Please cite:
I. S. Vlachos, M. D. Paraskevopoulou, D. Karagkouni, G. Georgakilas, T. Vergoulis, I. Kanellos, I-L. Anastasopoulos, S. Maniou, K. Karathanou, D. Kalfakakou, A. Fevgas, T. Dalamagas and A. G. Hatzigeorgiou. DIANA-TarBase v7.0: indexing more than half a million experimentally supported miRNA:mRNA interactions. Nucl. Acids Res. (2014)
Data Download:
DIANA-TarBase v7 is available for scientific non-profit and non-commercial use! Download TarBase v7 by following this link
|
||||||||
Related Pathways
Publication year
Prediction score
Filters
Selected:
Species: -
Regulation: -
Validation: -
Validated: -
Sources: -
Methods: -
Species: -
Regulation: -
Validation: -
Validated: -
Sources: -
Methods: -
Remove all 
Species
Method Type
Method
Regulation type
Validation type
Validated as
Source
Publication year
Prediction score
Loading... |
| Wait until the result set is completed... |
Gene name
miRNA name
Methods
Pred.Score
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr12:9093296-9093334 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:9094250-9094275 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:9094303-9094336 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:9098943-9099001 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:9102103-9102131 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:53362947-53362989 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr6:53409669-53409697 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:16147173-16147204 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr18:9536361-9536388 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr18:9537459-9537486 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SCMH1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:41540951-41540986 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
DHX33 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:5345762-5345808 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CD44 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:35251355-35251367 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
HCCS (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chrX:11141142-11141165 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
BAZ1B (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:72892500-72892534 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
RNF216 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:5661097-5661119 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
AKAP11 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr13:42895194-42895226 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr13:42876075-42876109 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr13:42891788-42891804 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
DVL2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:7131305-7131332 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
GCFC2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr2:75929443-75929463 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
TBPL1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
QPCTL (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:46207016-46207027 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:46207027-46207066 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NSMAF (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr8:59515852-59515872 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SYNRG (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:35878440-35878461 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr17:35937627-35937669 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
UPF1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:18976177-18976205 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
FUT8 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
BTBD7 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr14:93723595-93723616 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
BIRC3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:102192657-102192701 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
BCLAF1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:136590701-136590732 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
FAM136A (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
GAB2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:77926469-77926488 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:77926908-77926934 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:77931389-77931415 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:77937725-77937738 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:77961311-77961345 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
LASP1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:37075191-37075234 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr17:37075237-37075273 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ENPP4 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:46112326-46112360 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
KPNA6 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:32636864-32636909 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:32637697-32637710 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:32638931-32638961 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CDC27 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:45232083-45232111 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr17:45195126-45195176 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CAPN1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:64978877-64978922 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:64979203-64979230 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:64979304-64979315 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:64950629-64950654 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr11:64972211-64972236 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
VPS41 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:38765709-38765742 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CASP10 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr2:202083711-202083764 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr2:202086305-202086324 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
LAMP2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chrX:119570464-119570496 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chrX:119571687-119571699 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chrX:119571699-119571740 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chrX:119580239-119580269 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PSMB1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:170852702-170852722 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
YAF2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
LYPLA2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:24121387-24121408 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:24121521-24121550 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ST7 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:116863131-116863140 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr7:116863140-116863173 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PIK3C2A (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:17108960-17108987 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
TRIO (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr5:14508543-14508572 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
IFRD1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:112102090-112102114 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SLC30A9 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr4:42089319-42089348 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CDKL3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr5:133561532-133561565 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CCDC109B (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
UBR2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:42656086-42656106 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SLC39A9 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr14:69926850-69926889 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:69928022-69928052 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
HSF2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr6:122753507-122753528 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PSMC4 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:40478405-40478435 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
RNF19A (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr8:101300063-101300088 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MNAT1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
SPAG9 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:49043696-49043713 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
STRAP (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr12:16056238-16056259 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:16035462-16035491 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
GPRC5A (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr12:13065486-13065578 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:13067517-13067569 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr12:13070399-13070410 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ZDHHC6 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
NDUFS1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr2:206987642-206987671 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CIAPIN1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr16:57462592-57462632 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NCAPD2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr12:6619574-6619603 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MBTD1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:49255400-49255431 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ZFX (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chrX:24197863-24197880 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CDH1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr16:68868229-68868262 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MYCBP2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr13:77635800-77635829 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr13:77664345-77664387 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr13:77718598-77718638 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ANLN (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:36493371-36493380 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr7:36446008-36446025 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr7:36450217-36450238 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr7:36450730-36450754 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ADIPOR2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr12:1897510-1897537 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
TFPI (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr2:188382421-188382455 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MAT2B (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
MAP3K9 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr14:71196216-71196236 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:71189769-71189786 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:71190583-71190608 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:71190866-71190893 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:71190967-71190989 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr14:71192075-71192109 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
WIZ (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:15533025-15533051 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:15538912-15538950 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
IFNGR1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:137525623-137525640 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PGM3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:83874769-83874778 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
TIMP2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:76849156-76849195 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr17:76851477-76851506 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
VCL (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr10:75878079-75878099 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr10:75834512-75834543 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr10:75860826-75860856 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr10:75863583-75863607 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
RPUSD1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr16:835158-835185 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CX3CL1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr16:57417150-57417192 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ST3GAL1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr8:134470879-134470904 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NUP160 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:47826138-47826161 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SLC25A5 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chrX:118605155-118605175 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chrX:118603746-118603781 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chrX:118604344-118604374 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CSDE1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:115282473-115282507 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PTBP1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:808721-808763 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ITGA3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:48167160-48167198 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NSUN2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr5:6623326-6623363 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CCDC28A (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:139097216-139097254 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PHF20 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr20:34536363-34536385 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr20:34457360-34457396 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
RALA (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr7:39663231-39663273 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
Microarrays
NEGATIVE
INDIRECT
Tarbase 7.0
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
Western Blot
NEGATIVE
INDIRECT
Tarbase 7.0
SYPL1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr7:105731705-105731748 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr7:105752657-105752710 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
ADSS (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:244571869-244571908 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:244572707-244572735 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:244615126-244615169 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr1:244582222-244582262 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MARK4 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:45649145-45649174 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
PAFAH1B1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:2497196-2497242 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
AP2B1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:34051559-34051591 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
FUCA2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
chr6:143823598-143823616 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
EXTL3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr8:28575344-28575362 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NIPAL3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:24798984-24799013 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
BID (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr22:18218185-18218233 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NLRP2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:55493666-55493698 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:55494125-55494147 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SLC22A16 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr6:110777558-110777582 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SLC45A4 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr8:142231794-142231813 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr8:142264659-142264686 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
GRAMD1B (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr11:123493563-123493581 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
SNX11 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr17:46199582-46199623 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
CRLF1 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:18686420-18686449 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
NFIX (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr19:13205576-13205611 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:13207048-13207086 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:13209510-13209538 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
chr19:13184269-13184282 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MFAP3 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr5:153432583-153432600 (UNKNOWN)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
AK2 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:33474792-33474826 (3UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0
MTMR11 (hsa)
hsa-miR-522-5p
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
chr1:149908707-149908748 (5UTR)
We applied an unbiased sequencing-based systems approach to characterize miR-522, a member of the oncogenic primate-specific chromosome 19 miRN cluster, highly expressed in poorly differentiated cancers. To identify miRN targets, we sequenced full-length transcripts captured by a biotinylated miRN mimic. Within these targets, mostly noncanonical MREs were identified by sequencing RNase-resistant fragments.
IMPACT-Seq
POSITIVE
DIRECT
Tarbase 7.0