Deprecated!
This application is deprecated. Please use the latest version of the tool here.
Please cite:
I. S. Vlachos, M. D. Paraskevopoulou, D. Karagkouni, G. Georgakilas, T. Vergoulis, I. Kanellos, I-L. Anastasopoulos, S. Maniou, K. Karathanou, D. Kalfakakou, A. Fevgas, T. Dalamagas and A. G. Hatzigeorgiou. DIANA-TarBase v7.0: indexing more than half a million experimentally supported miRNA:mRNA interactions. Nucl. Acids Res. (2014)
Data Download:
DIANA-TarBase v7 is available for scientific non-profit and non-commercial use! Download TarBase v7 by following this link
|
||||||||
Related Pathways
Publication year
Prediction score
Filters
Selected:
Species: -
Regulation: -
Validation: -
Validated: -
Sources: -
Methods: -
Species: -
Regulation: -
Validation: -
Validated: -
Sources: -
Methods: -
Remove all
Species
Method Type
Method
Regulation type
Validation type
Validated as
Source
Publication year
Prediction score
Loading... |
Wait until the result set is completed... |
Gene name
miRNA name
Methods
Pred.Score
Olr758 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Thbd (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cldn8 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Mageh1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
2310047M10Rik (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tbp (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rps27a (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Egfr (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Sec61g (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Wdr19 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
THEGL (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cxcl2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ereg (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Hsd17b13 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Arsg (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Map3k14 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Gpr19 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Lrrc59 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Xcr1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Bfsp1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tefm (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Serpinf2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rap1gap2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Itpka (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Fsip1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Nhp2l1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
RGD1562178 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Foxred2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Usp43 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Glp2r (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Smyd1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cyp46a1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Myocd (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Hoxd9 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Adora2b (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Bbs5 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Utp23 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Scn9a (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Zfp786 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Pde4b (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Wdsub1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tuba4a (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cyp19a1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tpst2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Gfpt2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Hps4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Dao (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tmem19 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Gltp (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cabp1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Srsf9 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Nr2c1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ntn4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tomm40l (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
P2ry4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Efnb1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Fmo4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
2510003E04Rik (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Hgf (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tnn (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
RGD1304982 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Pcbd1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Traf2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tsen15 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Nrbf2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Bach2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rnf32 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Gpm6b (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tlr7 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Frmpd4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rbks (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Col18a1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Dohh (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ncln (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
RGD1309594 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Fmod (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Mepce (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Slc37a1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rsph1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Atp13a4 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Eif2d (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Pkdcc (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
C4bpb (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cpne5 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Dpp10 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Htr5b (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Phf1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Zbtb22 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Serpinb7 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Cdo1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ptcd1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Zfp394 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ltb (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Tnf (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Lnx2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Col11a2 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Ddr1 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Dcaf15 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Rpp21 (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0
Map3k7cl (rno)
rno-miR-128
-
Publication
Methods
Tissue
Cell line
Tested cell line
Exp. condition
Location
Method
Result
Regulation
Valid. type
Source
UNKNOWN
Ago2 immunoprecipitation followed by deep sequencing is a useful tool to identify novel miRN targets. Upregulation of neuronal miR-9, miR-125b, and miR-128 during myofibroblastic transition and the identified interaction with a wide range of chemokines and chemokine receptors suggest a prominent role of neuronal miRNs in the inflammatory response of HSC during fibrosis.
AGO-IP
POSITIVE
INDIRECT
Tarbase 7.0